Copper and Coronavirus, blood levels need to be tested. A possible way that Coronavirus is working in the body.
Emailed to Asim K. Duttaroy, Corresponding author of "Is copper beneficial for COVID-19 patients?" (Details at bottom.)
Greetings.
Shortness of breath and chest pain are common iron deficiency symptoms. When there isn't enough copper in the body, various transport chemicals cannot move iron around correctly. Bioavailable iron becomes insufficient, causing anemia and shortness of breath and chest pain, and eventually, the left ventricle will expand.
Some circumstances will allow high copper to interfere with iron homeostasis, as well, but I'm having a harder time documenting it as it seems. You probably know more about this than I do. But I've experienced it, which is why I have studied it.
(My copper homeostasis has been interfered with by the addition of a molecule called copper 2 Omeprazole complex, or Cu2(OM), causing symptoms which match fibromyalgia, and probably caused some fibromyalgia from other people who I've talked to who share the symptoms, and share the benefits of Epsom salts, and to which I have a cure, but now is not the time. Merely that I have personal experience that has driven correct questions to get useful answers which now seem to apply to Coronavirus.)
When zinc is deficient, usually extra copper is unable to leave the body correctly. The exceptions would be when there isn't sufficient copper in the first place, for reasons related or unrelated, or if the copper was insufficient because the zinc was being prioritized to remove it from the body somehow, which could be what coronavirus is doing if copper is low in the blood.
Of course, I haven't heard anyone ask where the zinc is going. We're putting in so much, where is it going? If we answer that question, we might be able to figure out how to get it going back to the immune system correctly.
Copper would be extremely beneficial if the reason the zinc is low in the immune system is because it is being prioritized to remove copper (versus other zinc antagonists) from the body, removing both from the system. Resultant insufficient copper, by causing an iron deficiency, would also cause shortness of breath, and chest pain.
It need not be that simple, but Occam's razor says that that's where we should start looking.
Has anybody checked the copper levels (and free copper versus bound copper of people with Coronavirus? I can't find any results. Likewise iron.
Free radical copper, such as proliferates in type 2 diabetes because the atp7a molecule isn't able to hold on to Copper correctly, which I believe is a major step in how diabetes is caused, but not the final one, is not bioavailable for all uses. Again, you probably know more about this than I do.
I postulate one of these two likelihoods. Either the zinc is being deflected from the immune systems of those suffering coronavirus severely, by getting rid of extra copper.
"Per A Review on Kombucha Tea [28], "It was concluded that essential minerals (Cu, Fe, Mn, Ni, and Zn) increased as a result of the metabolic activity of kombucha."
"Could Coronavirus increase the amount of copper or some other zinc antagonists in the same fashion?"
([28] A Review on Kombucha Tea—Microbiology, Composition, Fermentation, Beneficial Effects, Toxicity, and Tea FungusRasu Jayabalan, Radomir V. Malbaša, Eva S. Lončar, Jasmina S. VitasFirst published: 21 June 2014
Comprehensive Reviews in Food Science and Food Safety, Volume 13, Issue 4)
Quoting my website, JoREM
The other alternative is that insufficient or deficient zinc would have to otherwise be unable to get rid of copper, and there would be too much copper in the body and the brain. Although I find a lot less information on that, it can cause anemia under certain circumstances, and if this is the case was Coronavirus, then coronavirus offers those circumstances.
Under that alternative, adding copper to the bodies of coronavirus sufferers would be detrimental.
Although copper is necessary for the immune system, additional copper is liable to be detrimental even if it is at normal levels, two people with coronavirus, because the body will deflect even a little of the zinc of which it is in such short supply, and the immune system will suffer.
So, through simple lab tests, and the more complex lab test of bound versus chaperone and transported copper, you could easily prove whether copper is going to be beneficial or detrimental, and if it's slow, you can add that to your paper as well.
These ideas are on my website, which are copyright to me, ©Deborah Barges, under open access. If you find them useful, you should reference me for my ideas. If so, please let me know, so I can use it as credentials and get an ear for my research on fibromyalgia. It's frustrating having a cure, the name of the molecule, and nobody listening.
Sincerely,
Deborah Barges
---
Details:
Is copper beneficial for COVID-19 patients?
Syamal Raha, Rahul Mallick, [...], and Asim K. Duttaroy
Med Hypotheses. 2020 Sep; 142: 109814.
Published online 2020 May 5. doi: 10.1016/j.mehy.2020.109814
PMCID: PMC7199671
PMID: 32388476
-
Since then, I have read the admittedly dated
THE ANEMIA OF INFECTION. I.
HYPOFERREMIA, HYPERCUPREMIA, AND
ALTERATIONS IN PORPHYRIN METABOLISM IN
PATIENTS
By G. E. CARTWRIGHT, M. A. LAURITSEN, P. J. JONES, I. M. MERRILL,
AND M. M. WINTROBE
(From the Department of Medicine, University of Utah School of Medicine, Salt Lake City)
J Clin Invest. 1946;25(1):65-80. https://doi.org/10.1172/JCI101690
A pdf.
In it is concluded, as the third item, that they found hypoferremia, hypercupremia, higher protoporphyrin contained in the red blood cells, and coproporphyrinuria, and nornal levels of plasma bilirubin and total plasma proteins.
In the fifth item it was concluded that infection induced anemia corresponds to the anemia of iron deficiency in some ways, but is not helped by the same treatment.
And as the sixth item, that, copper, the vitamin B complex, vitamin C, "amigen," globin, cystine, methionine and crude liver did not help the anemia.
The search function did not find the word zinc anywhere in the document, so it looks like they did not try any copper antagonists to improve the infection induced anemia.
Increasing copper when there was already hypercupremia was not effective then, and it should be checked to see if the copper levels are high, low, or normal before it is attempted now. Lab results of blood from people actively having Coronavirus will show whether they have anemia, and whether it corresponds to the infection anemia seen in this document and as a result whether it will be safe to attempt increasing copper has a treatment.
Even if copper is low, the question is how it would be low when there is already a lack of zinc to remove it from the body. If coronavirus is telling the body to use the zinc to get rid of copper, adding more copper might or might not improve the situation. Adding more copper might simply allow coronavirus to tell the body to get rid of more zinc by getting rid of the copper. Testing using blood in vitro might be a step but it would not be a guarantee.
On the other hand, if that is happening, and the copper is still depleted after the person recovers, then adding copper would be a way to reduce what seems to be at this time Guillaim-Barre syndrome, but could be low copper demyelination, which mimics it.
I must assume that the people who presented this hypothesis had as little access to the blood of coronavirus victims as I do, because the test for copper where I live in 2019 was only $44, and would be an easy investment if they did have access.
Blood tests should be done before the hypothesis is tested, and even if copper is low, testing of this hypothesis should be done very, very carefully, to avoid having the zinc in the body be prioritized to removing the copper and away from the immune systems of people who currently are infected with coronavirus.
-----
©Deborah Barges July 2020 open access
